2010, ISBN: 9783642065590

[ED: Softcover], [PU: Springer / Springer Berlin Heidelberg / Springer, Berlin], Communication between cells via intercellular channels - gap junctions - appears essential to certain deve… More...

[ED: Softcover], [PU: Springer / Springer Berlin Heidelberg / Springer, Berlin], Communication between cells via intercellular channels - gap junctions - appears essential to certain developmental processes and appropriate organ function. Gap Junctions in Development and Disease aims to describe the molecular events underlying impaired development and disease. Beginning with a comprehensive review of various mouse and human genes encoding the channel-forming connexins, later chapters describe several connexin mutations associated with human diseases such as hereditary deafness and female infertility. Erroneous signaling mediated by the interaction of mutant connexins with other proteins, thought to be responsible for dysfunction of organs such as heart, muscle, brain, skin, lens, placenta, and endocrine tissue in both mice and men, is also addressed. Although the question of why some mutations in gap-junction proteins lead to specific phenotypes remains to be answered, the reviews in this book provide an intriguing insight into the future direction of this research field. 2010. xvii, 279 S. XVII, 279 p. 47 illus., 10 illus. in color. 235 mm Versandfertig in 6-10 Tagen, DE, [SC: 0.00], Neuware, gewerbliches Angebot, Offene Rechnung (Vorkasse vorbehalten)<

2010, ISBN: 3642065597

[EAN: 9783642065590], Neubuch, [SC: 0.0], [PU: Springer Berlin Heidelberg], MOUSE; MUTATION; PLACENTA; PROTEIN; PROTEINS; TISSUE; TRANSCRIPTION; CARDIACREGENERATION; BRAIN; CANCER; GENES;… More...

[EAN: 9783642065590], Neubuch, [SC: 0.0], [PU: Springer Berlin Heidelberg], MOUSE; MUTATION; PLACENTA; PROTEIN; PROTEINS; TISSUE; TRANSCRIPTION; CARDIACREGENERATION; BRAIN; CANCER; GENES; GENOME; INFERTILITY, Druck auf Anfrage Neuware - Printed after ordering - Communication between cells via intercellular channels - gap junctions - appears essential to certain developmental processes and appropriate organ function. Gap Junctions in Development and Disease aims to describe the molecular events underlying impaired development and disease. Beginning with a comprehensive review of various mouse and human genes encoding the channel-forming connexins, later chapters describe several connexin mutations associated with human diseases such as hereditary deafness and female infertility. Erroneous signaling mediated by the interaction of mutant connexins with other proteins, thought to be responsible for dysfunction of organs such as heart, muscle, brain, skin, lens, placenta, and endocrine tissue in both mice and men, is also addressed.Although the question of why some mutations in gap-junction proteins lead to specific phenotypes remains to be answered, the reviews in this book provide an intriguing insight into the future direction of this research field. 300 pp. Englisch, Books<

ISBN: 9783642065590

Communication between cells via intercellular channels - gap junctions - appears essential to certain developmental processes and appropriate organ function. Gap Junctions in Development … More...

Communication between cells via intercellular channels - gap junctions - appears essential to certain developmental processes and appropriate organ function. Gap Junctions in Development and Disease aims to describe the molecular events underlying impaired development and disease. Beginning with a comprehensive review of various mouse and human genes encoding the channel-forming connexins, later chapters describe several connexin mutations associated with human diseases such as hereditary deafness and female infertility. Erroneous signaling mediated by the interaction of mutant connexins with other proteins, thought to be responsible for dysfunction of organs such as heart, muscle, brain, skin, lens, placenta, and endocrine tissue in both mice and men, is also addressed. br / br / br / br /Although the question of why some mutations in gap-junction proteins lead to specific phenotypes remains to be answered, the reviews in this book provide an intriguing insight into the future direction of this research field. Engelse Boeken School studieboeken Alle school studieboeken Engelse Boeken > School studieboeken > Alle school studieboeken Paperback, [PU: Springer, Berlin/Heidelberg/New York, NY]<

ISBN: 9783642065590

Paperback, [PU: Springer-Verlag Berlin and Heidelberg GmbH & Co. KG], Communication between cells via intercellular channels - gap junctions - appears essential to certain developmental p… More...

Paperback, [PU: Springer-Verlag Berlin and Heidelberg GmbH & Co. KG], Communication between cells via intercellular channels - gap junctions - appears essential to certain developmental processes and appropriate organ function. Gap Junctions in Development and Disease aims to describe the molecular events underlying impaired development and disease. Beginning with a comprehensive review of various mouse and human genes encoding the channel-forming connexins, later chapters describe several connexin mutations associated with human diseases such as hereditary deafness and female infertility. Erroneous signaling mediated by the interaction of mutant connexins with other proteins, thought to be responsible for dysfunction of organs such as heart, muscle, brain, skin, lens, placenta, and endocrine tissue in both mice and men, is also addressed. Although the question of why some mutations in gap-junction proteins lead to specific phenotypes remains to be answered, the reviews in this book provide an intriguing insight into the future direction of this research field., Cellular Biology (cytology)<

ISBN: 9783642065590

Communication between cells via intercellular channels – gap junctions – appears essential to certain developmental processes and appropriate organ function. Gap Junctions in Development … More...

Communication between cells via intercellular channels – gap junctions – appears essential to certain developmental processes and appropriate organ function. Gap Junctions in Development and Disease aims to describe the molecular events underlying impaired development and disease. Beginning with a comprehensive review of various mouse and human genes encoding the channel-forming connexins, later chapters describe several connexin mutations associated with human diseases such as hereditary deafness and female infertility. Erroneous signaling mediated by the interaction of mutant connexins with other proteins, thought to be responsible for dysfunction of organs such as heart, muscle, brain, skin, lens, placenta, and endocrine tissue in both mice and men, is also addressed. Although the question of why some mutations in gap-junction proteins lead to specific phenotypes remains to be answered, the reviews in this book provide an intriguing insight into the future direction of this research field. Books > Life Sciences Soft cover, Springer Shop<