2011, ISBN: 9783639330144

[ED: Taschenbuch / Paperback], [PU: VDM Verlag Dr. Müller], Inflammation is a complex physiological response to pathogens. A prominent mediator of inflammation is the transcription factor… More...

[ED: Taschenbuch / Paperback], [PU: VDM Verlag Dr. Müller], Inflammation is a complex physiological response to pathogens. A prominent mediator of inflammation is the transcription factor NF- B, that acts as key transcriptional regulator of many genes coding for pro-inflammatory cytokines. Different signaling pathways activated by very diverse stimuli converge on NF- B, resulting in a regulatory network characterized by high complexity. Scope of this analysis is to provide a wider, systemic picture of such intricate signaling and interaction network: the NF- B pathway interactome. The study has been carried out following a workflow for gathering information from literature as well as from several pathway and protein interactions databases, and for integrating and analyzing existing data and the relative reconstructed representations and mathematically analyzable networks. The reconstruction of the NF- B interactome pursued with this approach provides a starting point for a general view of the architecture and for a deeper analysis and understanding of this complex regulatory system., DE, [SC: 0.00], Neuware, gewerbliches Angebot, 136, Selbstabholung und Barzahlung, PayPal, offene Rechnung, Banküberweisung, Interntationaler Versand<

2011, ISBN: 9783639330144

[ED: Taschenbuch / Paperback], [PU: VDM Verlag Dr. Müller], Inflammation is a complex physiological response to pathogens. A prominent mediator of inflammation is the transcription factor… More...

[ED: Taschenbuch / Paperback], [PU: VDM Verlag Dr. Müller], Inflammation is a complex physiological response to pathogens. A prominent mediator of inflammation is the transcription factor NF- B, that acts as key transcriptional regulator of many genes coding for pro-inflammatory cytokines. Different signaling pathways activated by very diverse stimuli converge on NF- B, resulting in a regulatory network characterized by high complexity. Scope of this analysis is to provide a wider, systemic picture of such intricate signaling and interaction network: the NF- B pathway interactome. The study has been carried out following a workflow for gathering information from literature as well as from several pathway and protein interactions databases, and for integrating and analyzing existing data and the relative reconstructed representations and mathematically analyzable networks. The reconstruction of the NF- B interactome pursued with this approach provides a starting point for a general view of the architecture and for a deeper analysis and understanding of this complex regulatory system., DE, [SC: 0.00], Neuware, gewerbliches Angebot, 136, Selbstabholung und Barzahlung, PayPal, offene Rechnung, Banküberweisung, Internationaler Versand<

ISBN: 9783639330144

[ED: Taschenbuch], [PU: VDM Verlag Dr. Müller e.K.], Neuware - Inflammation is a complex physiological response to pathogens. A prominent mediator of inflammation is the transcription fac… More...

[ED: Taschenbuch], [PU: VDM Verlag Dr. Müller e.K.], Neuware - Inflammation is a complex physiological response to pathogens. A prominent mediator of inflammation is the transcription factor NF- B, that acts as key transcriptional regulator of many genes coding for pro-inflammatory cytokines. Different signaling pathways activated by very diverse stimuli converge on NF- B, resulting in a regulatory network characterized by high complexity. Scope of this analysis is to provide a wider, systemic picture of such intricate signaling and interaction network: the NF- B pathway interactome. The study has been carried out following a workflow for gathering information from literature as well as from several pathway and protein interactions databases, and for integrating and analyzing existing data and the relative reconstructed representations and mathematically analyzable networks. The reconstruction of the NF- B interactome pursued with this approach provides a starting point for a general view of the architecture and for a deeper analysis and understanding of this complex regulatory system., [SC: 0.00], Neuware, gewerbliches Angebot, 220x150x8 mm, [GW: 219g]<

ISBN: 9783639330144

Inflammation is a complex physiological response to pathogens. A prominent mediator of inflammation is the transcription factor NF- B, that acts as key transcriptional regulator of many g… More...

Inflammation is a complex physiological response to pathogens. A prominent mediator of inflammation is the transcription factor NF- B, that acts as key transcriptional regulator of many genes coding for pro-inflammatory cytokines. Different signaling pathways activated by very diverse stimuli converge on NF- B, resulting in a regulatory network characterized by high complexity. Scope of this analysis is to provide a wider, systemic picture of such intricate signaling and interaction network: the NF- B pathway interactome. The study has been carried out following a workflow for gathering information from literature as well as from several pathway and protein interactions databases, and for integrating and analyzing existing data and the relative reconstructed representations and mathematically analyzable networks. The reconstruction of the NF- B interactome pursued with this approach provides a starting point for a general view of the architecture and for a deeper analysis and understanding of this complex regulatory system. Bücher / Naturwissenschaften, Medizin, Informatik & Technik / Biologie / Mikrobiologie, [PU: VDM Verlag Dr. Müller, Saarbrücken]<

2011, ISBN: 3639330145

Hardcover

Reconstructing the interactome involved in the activation of the master regulator of inflammation: a network and systems biology approach - Buch, gebundene Ausgabe, 136 S., Beilagen: Pape… More...

Reconstructing the interactome involved in the activation of the master regulator of inflammation: a network and systems biology approach - Buch, gebundene Ausgabe, 136 S., Beilagen: Paperback, Erschienen: 2011 VDM Verlag Dr. Müller e.K., [PU: VDM Verlag Dr. Müller, Saarbrücken]<